Current/Future Research
- Recent research has shown that damage to the medial vestibulospinal tract alters vestibular evoked myogenic potential in the sternocleidomastoid muscle (SCM), which are involved in head rotation. The vestibular evoked myogenic potential is an assessment of the sacculo-collic reflex and a test of function in otolithic organs. Also, lesions to the tract impair ascending efferent fiber signaling, which led to nystagmus.
- There is has also been recent research to determine if there is a difference in vestibulospinal function when there is damage to the superior vestibular nerve as opposed to the inferior vestibular nerve and vice versa. They defined vestibulospinal function by ability to have proper posture, as well as by self reported dizziness. The results were determined by using the Sensory Organization Test (SOT) of the computerized dynamic posturography (CDP) as well as the dizziness handicap inventory (DHI). It was determined that subjects with damaged inferior spinal nerve performed worse on the posture test than the control group, but performed better that patients with superior vestibulo nerve damage. With this they determined that the superior vestibular nerve plays a larger in balance than the inferior vestibulo nerve but that they both play a role. In terms of the DHI is was concluded that there was no difference between the patients with the two different impairments.
- Vestibular compensation after unilateral or bilateral vestibular system damage can be accomplished by sensory addition and sensory substitution. Sensory substitution occurs when any remaining vestibular function, vision, or light touch of a stable surface substitute for the lost function. Postural sway and gait ataxia can be reduced by augmenting sensory information for balance control. Recent research has shown that as little as 100 grams of light touch of a fingertip can provide enough sensory reference to reduce sway and ataxia during gait.
Read more about this topic: Vestibulospinal Tract
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