ROS-directed Cancer Chemotherapeutics
Recent research demonstrates that redox dysregulation originating from metabolic alterations and dependence on mitogenic and survival signaling through ROS represents a specific vulnerability of malignant cells that can be selectively targeted by pro- and antioxidant redox chemotherapeutics.
Read more about this topic: Reactive Oxygen Species
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—Ishmael Reed (b. 1938)
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